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Xu, Haodong
One or more keywords matched the following items that are connected to Xu, Haodong
Item TypeName
Concept Myocytes, Cardiac
Academic Article Elevated MCU Expression by CaMKIIdB Limits Pathological Cardiac Remodeling.
Academic Article A small-molecule LF3 abrogates ß-catenin/TCF4-mediated suppression of NaV1.5 expression in HL-1 cardiomyocytes.
Academic Article Enhancement of ß-catenin/T-cell factor 4 signaling causes susceptibility to cardiac arrhythmia by suppressing NaV1.5 expression in mice.
Academic Article Opposing roles of TCF7/LEF1 and TCF7L2 in cyclin D2 and Bmp4 expression and cardiomyocyte cell cycle control during late heart development.
Academic Article Activation of Wnt/ß-catenin signaling by hydrogen peroxide transcriptionally inhibits NaV1.5 expression.
Academic Article APC controls asymmetric Wnt/ß-catenin signaling and cardiomyocyte proliferation gradient in the heart.
Academic Article Dynamic monitoring of oxidative DNA double-strand break and repair in cardiomyocytes.
Academic Article Deletion of FoxO1 leads to shortening of QRS by increasing Na(+) channel activity through enhanced expression of both cardiac NaV1.5 and ß3 subunit.
Academic Article Activation of extracellular signal-regulated kinase 5 reduces cardiac apoptosis and dysfunction via inhibition of a phosphodiesterase 3A/inducible cAMP early repressor feedback loop.
Academic Article Role of Ca2+/calmodulin-stimulated cyclic nucleotide phosphodiesterase 1 in mediating cardiomyocyte hypertrophy.
Academic Article Reactive oxygen species-induced activation of p90 ribosomal S6 kinase prolongs cardiac repolarization through inhibiting outward K+ channel activity.
Academic Article Fatty Acids Enhance the Maturation of Cardiomyocytes Derived from Human Pluripotent Stem Cells.
Academic Article Targeted deletion of Kv4.2 eliminates I(to,f) and results in electrical and molecular remodeling, with no evidence of ventricular hypertrophy or myocardial dysfunction.
Academic Article Reactive oxygen species suppress cardiac NaV1.5 expression through Foxo1.
Academic Article Transgenic expression of fatty acid transport protein 1 in the heart causes lipotoxic cardiomyopathy.
Academic Article Propofol Provides Cardiac Protection by Suppressing the Proteasome Degradation of Caveolin-3 in Ischemic/Reperfused Rat Hearts.
Academic Article A positive feedback loop of phosphodiesterase 3 (PDE3) and inducible cAMP early repressor (ICER) leads to cardiomyocyte apoptosis.
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  • Myocytes Cardiac